“Underneath that, there’s often an infection with Mycoplasma. We still don’t know if it’s really transferred with the same bite or if the people had it all along and become symptomatic when the immune system is suppressed by the spirochetes,” Dr. Klinghardt says.
Other than the co-infections, there is what I call the “opportunistic infections.” The combined effect of the initial infection is an immune suppressive effect, and then the patient becomes vulnerable to all sorts of other things. The most common things people contract early on in the course if the illness are different forms of parasites, such as protozoa; Babesia itself being one of them.
There is Giardia, amoebas, Trichomonas, malaria, and different forms of infections that aren’t labeled yet. There is a new one, called FL1953. Stephen Fry discovered that. It’s a protozoan organism that’s causing severe fatigue and illness in chronically ill people. It’s almost always present in a patient with Lyme disease.
And then we find a lot of worms in people. They may be microscopic and they may be macroscopic. That means they may be visible in the stool or they may not be visible.”
Common Presentations of Lyme Disease
The most simple presentations are the orthopedic forms of Lyme disease as they’re typically more superficial, affecting the larger joints. Interestingly, Dr. Klinghardt injects ozone into the joint in these cases, which he claims is:
“100 percent effective if the Lyme disease or… whatever the microbe is, lives in the joint and is confined to the joint space. Simply putting ozone in the joint will kill the spirochetes, and often with one, sometimes two treatments, make the joint completely pain free. That’s one form of the expression of Lyme.”
When the microbes and the associated immune reactions are situated in the connective tissue, the infection presents as a “vague, dispersed pain,” which oftentimes ends up being labeled as fibromyalgia by conventional doctors. The immunological expression of Lyme covers a wide variety of immune system disorders, typically with some aspect of autoimmunity. Dr. Klinghardt says:
“I did my thesis in 1976 on autoimmune diseases and how the autonomic nervous system interacts with the immune system. We found then that the determining factor of the outcome of an autoimmune disease was the presence of microbes that were catastrophically unresponsive to antibiotics.
Any autoimmune disease, including rheumatoid arthritis, we suspect has an underlying level of Lyme disease that needs to be treated appropriately before the patient has a chance to recover from the illness.”